<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0535-5133</journal-id>
<journal-title><![CDATA[Investigación Clínica]]></journal-title>
<abbrev-journal-title><![CDATA[Invest. clín]]></abbrev-journal-title>
<issn>0535-5133</issn>
<publisher>
<publisher-name><![CDATA[Instituto de Investigaciones Clínicas "Dr. Américo Negrette", Facultad de Medicina, Universidad del Zulia]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0535-51332023000300379</article-id>
<article-id pub-id-type="doi">10.54817/ic.v64n3a10</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Transición epitelio - mesenquima y cáncer.]]></article-title>
<article-title xml:lang="en"><![CDATA[Epithelial-mesenchymal transition and cancer.]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Arvelo]]></surname>
<given-names><![CDATA[Francisco]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Sojo]]></surname>
<given-names><![CDATA[Felipe]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Fundación Instituto de Estudios Avanzados-IDEA Área Salud ]]></institution>
<addr-line><![CDATA[Caracas ]]></addr-line>
<country>Venezuela</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad Central de Venezuela Instituto de Biología Experimental Laboratorio de Cultivo de Tejidos y Biología de Tumores]]></institution>
<addr-line><![CDATA[Caracas ]]></addr-line>
<country>Venezuela</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>09</month>
<year>2023</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>09</month>
<year>2023</year>
</pub-date>
<volume>64</volume>
<numero>3</numero>
<fpage>379</fpage>
<lpage>404</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://ve.scielo.org/scielo.php?script=sci_arttext&amp;pid=S0535-51332023000300379&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://ve.scielo.org/scielo.php?script=sci_abstract&amp;pid=S0535-51332023000300379&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://ve.scielo.org/scielo.php?script=sci_pdf&amp;pid=S0535-51332023000300379&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen La migración e invasión de células cancerosas son componentes claves de la enfermedad metastásica, que es la principal causa de muerte en pacientes con cáncer. La transición epitelio-mesenquima (TEM) y la transición mesenquima-epitelio (TME) son una vía implicada en la metástasis del cáncer. Este proceso comprende la degradación de las uniones célula-célula y célula- matríz extracelular, y la subsecuente pérdida de la regulación de proteínas de unión como la caderina-E, por lo que las células experimentan una reorganización del citoesqueleto. Estas alteraciones están asociadas con un cambio de la forma celular, de una morfología epitelial a una mesenquimatosa. La comprensión de la base molecular y celular de la TEM y de la TME, proporciona conocimientos fundamentales sobre la etiología del cáncer, que pueden conducir a nuevas estrategias terapéuticas. En esta revisión, discutimos algunos de los mecanismos reguladores y el papel patológico de la plasticidad epitelio-mesénquima, con un enfoque en los conocimientos sobre la complejidad y la dinámica de este fenómeno en el cáncer.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Cancer cell migration and invasion are critical components of metastatic disease, the leading cause of death in cancer patients. The epithelium-mesenchyme-transition (EMT) and mesenchyme-epithelium-transition (MET) are pathways involved in cancer metastasis. This process involves the degradation of cell-cell and cell-extracellular matrix junctions and the subsequent loss of regulation of binding proteins such as E-cadherin. Cells undergo a reorganization of the cytoskeleton. These alterations are associated with a change in cell shape from epithelial to mesenchymal morphology. Understanding EMT and MET&#8217;s molecular and cellular basis provides fundamental insights into cancer etiology and may lead to new therapeutic strategies. In this review, we discuss some of the regulatory mechanisms and pathological role of epithelial-mesenchymal plasticity, focusing on the knowledge about the complexity and dynamics of this phenomenon in cancer.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[cáncer]]></kwd>
<kwd lng="es"><![CDATA[epitelio]]></kwd>
<kwd lng="es"><![CDATA[caderina]]></kwd>
<kwd lng="es"><![CDATA[plasticidad]]></kwd>
<kwd lng="es"><![CDATA[transición epitelio-mesenquima]]></kwd>
<kwd lng="en"><![CDATA[cancer]]></kwd>
<kwd lng="en"><![CDATA[epithelium]]></kwd>
<kwd lng="en"><![CDATA[cadherin]]></kwd>
<kwd lng="en"><![CDATA[plasticity]]></kwd>
<kwd lng="en"><![CDATA[epithelial-mesenchymal transition]]></kwd>
</kwd-group>
</article-meta>
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