<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2960-2467</journal-id>
<journal-title><![CDATA[e-Revista Multidisciplinaria del Saber]]></journal-title>
<abbrev-journal-title><![CDATA[e-Rev. Multidiscip. Saber]]></abbrev-journal-title>
<issn>2960-2467</issn>
<publisher>
<publisher-name><![CDATA[Centro de Investigación en Tecnologías de Salud y Ambiente (CITSA)]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2960-24672026000100405</article-id>
<article-id pub-id-type="doi">10.61286/e-rms.v4i.371</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Arquitectura genética y neurobiológica de los trastornos del neurodesarrollo: una revisión integrativa de TDAH, TEA y discapacidad intelectual]]></article-title>
<article-title xml:lang="en"><![CDATA[Genetic and neurobiological architecture of neurodevelopmental disorders: An integrative review of ADHD, ASD, and intellectual disability]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Mesta Ventura]]></surname>
<given-names><![CDATA[Carlos Rodrigo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Gómez Canicela]]></surname>
<given-names><![CDATA[Franklin]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Continental  ]]></institution>
<addr-line><![CDATA[Huancayo ]]></addr-line>
<country>Perú</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2026</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2026</year>
</pub-date>
<volume>4</volume>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://ve.scielo.org/scielo.php?script=sci_arttext&amp;pid=S2960-24672026000100405&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://ve.scielo.org/scielo.php?script=sci_abstract&amp;pid=S2960-24672026000100405&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://ve.scielo.org/scielo.php?script=sci_pdf&amp;pid=S2960-24672026000100405&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen Los trastornos del neurodesarrollo constituyen condiciones heterogéneas caracterizadas por alteraciones tempranas del desarrollo cerebral, entre las que destacan el trastorno por déficit de atención con hiperactividad (TDAH), el trastorno del espectro autista (TEA), y la discapacidad intelectual. El objetivo del presente trabajo fue integrar los principales avances genéticos y neurobiológicos asociados a estos trastornos, con el fin de ofrecer una visión actualizada de sus mecanismos etiológicos. Para ello, se realizó una revisión narrativa no sistemática de la literatura científica publicada entre los años 2000 y enero de 2026, empleando bases de datos especializadas como PubMed, Scopus, Web of Science, PyscINFO y ScienceDirect, con búsquedas complementarias en Google Scholar. La evidencia disponible muestra que estos trastornos presentan una arquitectura etiológica compleja, caracterizada por la interacción de factores genéticos, epigenéticos, neurobiológicos y ambientales que afectan procesos fundamentales del neurodesarrollo, tales como la sinaptogénesis, la plasticidad neuronal y la organización funcional de las redes cerebrales. En conjunto, los hallazgos revisados sugieren que la comprensión de estos trastornos requiere un enfoque integrador que articule los aportes de la genética, la neurobiología y la neuropsicología, lo que permite fortalecer el desarrollo de intervenciones profesionales basadas en evidencia.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Neurodevelopmental disorders constitute a heterogeneous group of conditions characterized by early alterations in brain development, among which attention-deficit/hyperactivity disorder (ADHD), autism spectrum disorder (ASD), and intellectual disability are particularly prominent. The objective of the present study was to integrate the main genetic and neurobiological advances associated with these disorders in order to provide an updated overview of their etiological mechanisms. To this end, a non-systematic narrative review of the scientific literature published between 2000 and January 2026 was conducted, using specialized databases such as PubMed, Scopus, Web of Science, PsycINFO, and ScienceDirect, with complementary searches in Google Scholar. The available evidence indicates that these disorders present a complex etiological architecture characterized by the interaction of genetic, epigenetic, neurobiological, and environmental factors that affect fundamental neurodevelopmental processes such as synaptogenesis, neuronal plasticity, and the functional organization of brain networks. Overall, the findings reviewed suggest that understanding these disorders requires an integrative approach that articulates contributions from genetics, neurobiology, and neuropsychology, thereby supporting the development of evidence-based professional interventions.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[trastornos del neurodesarrollo]]></kwd>
<kwd lng="es"><![CDATA[trastorno del espectro autista]]></kwd>
<kwd lng="es"><![CDATA[TDAH]]></kwd>
<kwd lng="es"><![CDATA[discapacidad intelectual]]></kwd>
<kwd lng="es"><![CDATA[redes neuronales]]></kwd>
<kwd lng="en"><![CDATA[neurodevelopmental disorders]]></kwd>
<kwd lng="en"><![CDATA[autism spectrum disorder]]></kwd>
<kwd lng="en"><![CDATA[attention-deficit/hyperactivity disorder]]></kwd>
<kwd lng="en"><![CDATA[Intellectual disability]]></kwd>
<kwd lng="en"><![CDATA[neural networks]]></kwd>
</kwd-group>
</article-meta>
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